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Hair Loss Industry Exposed - How I reversed my hair loss after 10 years with male pattern baldness! — Transcript

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0:00If you have hair loss, this is a video
0:02you need to watch. So, settle in, get a
0:06cup of coffee, feel relaxed. This is
0:08going to be a long presentation, but at
0:11the end of it, what I want to do is
0:14really get to the root cause of what is
0:16actually causing hair loss in men. I've
0:19spent the last 10 years researching
0:22this, going deep, and I think I've
0:24pretty much figured out how to stop hair
0:26loss for myself and how to regrow it to
0:29a certain extent. And this is basically
0:32the video that shows you how I've done
0:34it and why I've done it that way. And in
0:38this presentation, I'm going to go
0:39through about 50 different studies, and
0:41it's all going to link together in this
0:43kind of mind map uh that shows you what
0:47really causes hair loss in men. So,
0:50settle in. This is going to be a long
0:52one. You're going to absolutely love it.
0:54If you have hair loss, this is just an
0:56absolute mustwatch. Right. So, a little
0:59bit about me. At a young age, I had
1:02ultraaggressive hair loss. Right? I can
1:04still remember 17 years old. But I'm in
1:06school and my friend kind of cheekily
1:09told me that I had a receding hairline.
1:11And I was so naive at that point. I
1:13didn't even know what a receding
1:14hairline was. I didn't know that it was
1:17that was the beginning of male pattern
1:19baldness. I literally had no idea. But
1:22that's how young I was. So from about 17
1:26to 26, my hair loss aggressively
1:29continued to thin and recede. Um, you
1:32can see
1:34this photo, you know, I've I've got such
1:36a deep receded hairline. It's so thin on
1:38top. Uh, this hairline, this photo I
1:41basically just given up at that point.
1:43It was so thin and it was so receded
1:45that, you know, I'd basically just given
1:47up and shaved it off.
1:50And just for some context, basically all
1:52the men in my family are bold. My dad,
1:55uncle, cousin, both grandfathers. you
1:58know, it I was in real deep trouble when
2:00it came to my hair. Um, I was definitely
2:03on the genetic pathway to being bald by
2:06the age I am now. But I still I had this
2:09niggling feeling that the real cause of
2:12hair loss just wasn't being discussed,
2:14at least in the mainstream. And I I just
2:17couldn't I couldn't take this idea of
2:19the mainstream narrative that it's just
2:21genetics, it's just hormones at face
2:24value. the rebel and the engineer in me
2:28uh because I have a master's degree in
2:30chemical engineering. I just wanted
2:32deeper answers. I just wasn't satisfied.
2:38So, uh this is this is quite important
2:42and
2:43it's it's going to be a theme in this
2:45presentation to be honest. Um the hair
2:47loss industry, they have a secret. I was
2:51I was warned not to make this video and
2:53I'll get on to that a little bit later
2:55on. Uh I know this is going to be
2:57extremely controversial. I know this is
3:00going to make a lot of people upset and
3:02and angry and angry with me. But after
3:0610 years of researching hair loss,
3:08literally 10 years um with a master's
3:11degree in chemical engineering
3:12researching hair loss full-time, the
3:14following presentation is simply a
3:16collection of evidence that I found.
3:20So for many years, nothing worked for
3:23me. My hair kept getting worse. But when
3:25I realized when I had this insight that
3:28the hair loss industry is built around
3:31this idea of a customer for life. So
3:33remember this. remember this idea, a
3:35customer of life. Um, that's when I
3:38started realizing where I was going
3:41wrong. So, most importantly, in this
3:43video, I'll present my research, but
3:45it's it's up to you. It's really up to
3:47you to form your own opinion. I'm not
3:48I'm not telling you anything. I'm
3:50presenting the research that I found and
3:52what worked for me after 10 years of
3:54trying. So, check all the studies for
3:57yourself. Form your own conclusion. I'll
4:00I'll be linking all the studies. Um, and
4:02I'll also link to this presentation
4:04board so you can go and go through it
4:06yourself step by step. Take your time.
4:08Go through, look at every study. This
4:10isn't medical advice. This is just me
4:13presenting the evidence that I found.
4:16And further to that, unfortunately, most
4:19people have already just given up on
4:21their hair, right? They just say, "Oh,
4:22it's genetics. There's nothing you can
4:24do." And honestly, if if that's you, if
4:27if you think that is you, then um just
4:29please go ahead and close this video
4:31because this this presentation isn't for
4:33you, right?
4:35Okay. So, let's let's get into the meat
4:37of this presentation. What really causes
4:40hair loss in men? So, first up, a bit of
4:43context. The hair follicle is a mini
4:46organ. It's it's an an organ. um and as
4:50such it needs oxygen and it needs
4:53nutrients and it needs these minerals to
4:55survive right so think about the hair
4:58follicle in the context of being an
5:00organ because this is going to be super
5:01important going forward so as you can
5:04see blood flow arrives to the hair
5:06follicle bulb this this area here uh the
5:09dermal pillar it's also called via a
5:12network of blood vessels. So keep in
5:15mind this structure. Um this this really
5:18is an organ. It's got the erectile pilli
5:20muscle. It's got the sebaceous gland.
5:22And this is a this is a delicate complex
5:25uh organ.
5:28So hair follicle miniaturization is a
5:30hallmark of androgenetic alipcia which
5:33basically means when a dermatologist or
5:36a doctor goes to diagnose
5:39androgenetic alipcia male pattern
5:41baldness. you know what I had um they
5:44look for hair follicle miniaturization.
5:47So keep this in mind right this is this
5:49process where each successive hair
5:52growth cycle the hairs become thinner
5:54and wispier. It's it's not like with
5:57male pattern borders it's not like your
5:59hairs just fall out right it's that
6:01every cycle um they grow back slightly
6:06less strong slightly less thick and they
6:08grow slightly slower as well. So just
6:12keep this in mind as we go forward.
6:16So we know that balding scalps have
6:18lower blood flow. We know from multiple
6:20studies now that boarding scalps are
6:22deficient in blood flow compared to
6:24scalps with healthy hair. In fact, the
6:27studies show that balding scalps have
6:30about 2.6 times lower blood flow than
6:33scalps without hair loss. And that's a,
6:36you know, that's a massive difference.
6:37It's a very significant difference. So
6:40you can see this study here. SPF stands
6:42for subcutaneous blood flow and that was
6:452.6 times lower.
6:48So we're going to kind of go through
6:50quite a few points. You have to keep it
6:52in mind and keep the context in mind for
6:54the entire presentation.
6:56So let's move on to the next step.
7:00The studies now show us that balding
7:03scalps are significantly thinner and
7:05harder than scalps with healthy hair. So
7:08a 2021 study out Japan compared men with
7:11various stages of AA androgenetic
7:14alipcia keep that in mind to
7:16non-boarding men and on average the
7:18scalp of the boarding men was 5.9 mm
7:20thick compared to 7.8 mm in the
7:23controls. So that is a 25% difference.
7:27You know it's it's not something subtle
7:29that we're talking about here. It's a
7:31it's a noticeable real difference in the
7:33thickness of the scalp. And you can see
7:35from this study that, you know, they
7:38took the same spot on the scalp and it
7:42was significantly thinner in the
7:43boarding men. And just to sort of back
7:46up this point, you know, the thickness
7:48of the hypodermis and the entire scalp
7:50was significantly thinner in the AGA
7:53group than in the control group. You
7:56know, in conclusion, the study
7:57demonstrated that the thickness of the
7:59whole scalp and the hypodermis is lesser
8:00and the depth of the hair hair follicles
8:02is shallower in the AGA group.
8:05significantly shallower. And another
8:07study, the AJ group, showed reduced
8:10thickness of the entire scalp.
8:14So that's obviously been backed up by
8:16multiple studies. Um, and just as a side
8:20note, they also found that classic
8:22diminishment in blood flow um moving
8:24through the scalp in the men with the
8:25AGA. Then there was another study that
8:28found that Aga is associated with
8:30increased scalp hardness.
8:34So
8:36the occurrence and severity of Aga was
8:39positively correlated with scalp
8:40hardness in men. And this chart's really
8:43interesting because you can see the
8:45various spots on the scalp. And here you
8:48can see the different u grades. So the
8:51black line which is a little bit harder
8:53to see. The black line is basically no
8:57male pattern borders. Um the
9:01the green line is a little bit of hair
9:03loss and the red line is a lot of hair
9:05loss. So you can see in this spot MF2
9:08and MV that's where the biggest
9:10difference is and that's where men
9:12typically bald first. So it's what it's
9:15saying is the scalp is hardest in these
9:18areas MF1, MF2 and MV. And that's where
9:22the biggest difference is between the
9:23non-boarding and the boarding scalps.
9:27So, in reality, you don't really need a
9:29study to tell you this. Just look up
9:32close at any balding scalp and you'll
9:33see that scalp is thin and hard and
9:35leathery and it's been basically
9:38constantly compressed against the skull.
9:43So, what causes the pattern in male
9:46pattern baldness? We call it male
9:48pattern baldness for a reason. It's
9:50because it always takes place in this
9:51characteristic pattern and we've even
9:54given it a name and it's called the
9:55Hamilton Norwood scale of hair loss and
9:57you can see 1 through 7 how the hairline
10:00recedes and the pattern that it takes
10:02place in.
10:04So up until now scientists literally
10:08they had no explanation for why hair
10:11loss takes place in this pattern. The
10:13scientists simply they couldn't explain
10:15it. Right? And that was until there was
10:17a groundbreaking study that came along
10:19and it looked at the human scalp. So we
10:22can see here they looked at the scalp
10:26and tension across the scalp based on
10:28the shape of the skull and where the
10:32scalp was fixed. So at the front and the
10:35sides and the back. And what they found
10:37was this pattern where you can see the
10:40higher tension is sorry the higher
10:43tension is is here these shades. So at
10:46the front and on this crown area and the
10:48lower tension you can see the darker
10:51blue shades here. And what they found
10:54was
10:55absolutely incredible. This is the
10:58pattern of male pattern borders. So this
11:00is this typical norm Hamilton Norwood
11:03scale of hair loss. And you can see they
11:06look pretty much exactly the same. And
11:10the researchers found that this the
11:12chance of this being an actual
11:13coincidence is less than one in 1,000.
11:15So this overlap between the tension and
11:18the pattern of male pattern baldness is
11:21basically there's less than one in 1,000
11:24chance of that being coincidence. What
11:26they concluded was the elastic
11:28deformation of the scalp and AGA
11:30patterning clearly identifies mechanical
11:33stress as an active factor in AGA.
11:37So essentially the tension on the scalp
11:40is an active factor in andrew in your
11:44propensity to lose hair in that area.
11:47These this area at the front is high
11:50tension. This area in the front loes
11:53hair first. Same with the crown. High
11:55tension loses hair first.
11:58So let's go on to the next step because
12:00this is going to explain everything a
12:02little bit more.
12:06tension, as you can imagine, compresses
12:08the dermal layer. Right? Imagine the
12:10scalp tissue being pulled constantly
12:13tight over and against the skull. But
12:16what what do you think would happen over
12:17time? Right? I'd imagine that that that
12:19tissue would get, you know, compressed
12:22as it's being is being pulled that way
12:24and it's being pulled against the scalp
12:25and it gets compressed like that. And
12:28you can imagine this dermal layer, this
12:31delicate hair follicle with the um the
12:34hypodermis, the dermis, and the
12:36epidermis.
12:38You know, it's it's sort of like if you
12:40were to walk around barefoot for a year,
12:41the soles of your feet, they'd respond
12:44to that stimulus and they'd harden up,
12:46right? And this compression and this
12:49hardening is exactly what we see like we
12:52just talked about in the last in the
12:54last box, right? The scalp is harder and
12:58it's thinner than scalp with hair.
13:02And as you can probably imagine, a thin,
13:04hard, and a stiff scalp is much less
13:07vascular. And it makes it much more
13:09difficult for the delicate hair follicle
13:11bulb to actually survive, right? Imagine
13:14this delicate mini organ of the hair
13:17follicle with all those complex parts to
13:20it being squeezed and being contracted
13:23upon.
13:24So, we don't know exactly what causes
13:27this tension in the first place and why
13:29it happens, but clearly genetics is part
13:33of the story, and there's also clearly
13:36some lifestyle and environmental factors
13:38going into it.
13:40So, as you can see here, the part of the
13:44scalp that balds first, that's clearly
13:46the part that's being compressed, right?
13:49But on the sides and the back where the
13:51scalp is more vertical, the dermal layer
13:53is not compressed. It is getting pulled
13:56and stretched but it's not compressed
13:58against the skull. So the hair remains
14:00thick and healthy. You can see it's the
14:03exact part where
14:06um it's it's stops being compressed that
14:08the hair can still grow.
14:14Right? So keep that in mind. We're
14:15moving on to the next box.
14:18So guys, what about DHT?
14:21Most people know that DHT is clearly
14:24involved in male hair loss, right?
14:27Almost everyone knows about finasteride.
14:29It blocks DHT and that can stop hair
14:32loss very very effectively in men. But
14:34it's never really being clearly
14:36explained how that happens. You know, to
14:39me, it's just it's not an satisfactory
14:41explanation to say the hair follicle
14:43bulb is is sensitive to DHD. the hair
14:45follicle was just sensitive to DHT and
14:47that's why it falls out. You know, if
14:49that were true, blocking all the DHT,
14:52which is basically what happens with
14:54finasteride and dutastasteride, you
14:55block about 70 to 90% of the DHT with
14:58finasteride and dutasteride.
15:00Well, blocking it all would regrow all
15:02the hair on your head, but we know that
15:04it doesn't. In most cases, blocking DHT
15:07with finasteride, it stops the hair
15:09loss, but it regrows only a small amount
15:12of hair, right? It completely stops it,
15:15but it doesn't regrow that much. And
15:18this is because the real way that DHT
15:20causes hair loss is that DHT is a
15:23critical step in the pathway to
15:25fibrosis. So remember this word here
15:29because this is super important going
15:31forward with this presentation.
15:33Fibrosis.
15:35Women don't have high levels of DHT, so
15:37they don't get the same fibrosis as men.
15:40So let's look at how DHT in the body
15:44leads to fibrosis.
15:49So DHT and fibrosis, when muscles around
15:52the edge of your scalp pull on the
15:54scalp, they create that tension and then
15:55they create that scalp compression,
15:57right? But your body sees this chronic
16:01compression as a form of inflammation.
16:04It's kind of like an irritation that it
16:06wants to fix. It's like a an itch that
16:08needs to be scratched. It's it's
16:10something that needs to be, you know, it
16:12can't be ongoing because it's a form of
16:14inflammation. Your body wants to get in
16:16there and stop it. So to fight this
16:18perceived inflammation, your body sends
16:21DHT to that area of the scalp. DHT is
16:24actually an anti-inflammatory.
16:27And in the scalp, DHT signals the
16:29tissues to release a protein called
16:31transforming growth factor beta 1. And
16:34transforming growth factor B1 is known
16:36for its role in producing fibrosis.
16:38This protein tries to protect the scalp
16:41by creating fibrosis which is basically
16:43scar tissue made of collagen. Um it's
16:47it's an over production of disorganized
16:49collagen rather than these organized
16:51dermal layers where the hair bulb sits.
16:54So you can imagine that hair follicle
16:56mini organ. It sits within the layers of
16:58the the scalp the hypodermis the dermis
17:01and the epidermis. And when you get
17:04fibrosis, those layers become less
17:06distinct and the the sort of scar tissue
17:09that gets put down uh between those
17:12layers. The scar tissue is the body's
17:14way of quickly patching up wounds. It's
17:17tough. It kind of acts like this glue to
17:20resist the tension and the compression.
17:22But the fibrosis lacks the layered
17:24structure of normal scalp skin, which is
17:27crucial to the functioning of the hair
17:28follicle organ. Right? From the body's
17:31view, it's a win, right? because the
17:32tough scars resist the pulling and that
17:35stops the inflammation. But it causes
17:37this side effect, right? The fibrosis
17:40squeezes the hair follicle structure,
17:42leaving less room for the hair to grow
17:43and it blocks the blood vessels from
17:46delivering nutrients and oxygen. So in
17:48short, DHT is not actually attacking the
17:51hair follicle itself. It's responding to
17:53scalp tension and inflammation. And the
17:56result is the creation of fibrosis which
17:59you can imagine like a kind of scar
18:00tissue which makes the hair follicles
18:02miniaturaturize with each successive
18:05cycle. So we know this from the studies
18:08or the studies back up this idea.
18:11Follicular microinflammation plays an
18:13integral role in the pathogenesis of
18:15androgenetic alopecia. So over time
18:18thickening of parapholicular sheath
18:20takes place due to increased deposition
18:22of collagen resulting in marked
18:24parapholicular fibrosis and sometimes
18:27ends in the complete destruction of the
18:28affected hair follicles. So you can
18:31imagine this fibrosis basically
18:32strangling the hair follicle. And that's
18:35why after each successive hair growth
18:38cycle, the hair follicles become thinner
18:40and wispier and they grow less quickly
18:42because it's like they're being
18:44strangled both in terms of the blood
18:46supply and the growth space that the
18:48hair follicle has. And we have this
18:50quick animation that you can see. So the
18:53ga sits over the scalp. is being pulled
18:56tight and the dermal layers are attached
18:59intimately to the ga.
19:02So the hair follicle bulbs
19:05they're getting stretched and as a
19:07result the the dermal layer gets
19:09compressed
19:12that leads to the scalp hardening and
19:14thinning that we see in balding scalps
19:17and that inflammation leads to the
19:19creation of fibrosis via this DHT and
19:23transforming growth factor B21 pathway.
19:26that fibrosis sets in around the hair
19:29follicles and that leads them to
19:31miniaturaturize over each successive
19:33cycle.
19:36Okay, so hopefully this is all making
19:38sense so far. We've got
19:43the hair follicle bulb, then we have the
19:45lower blood flow, then we have the
19:46scalps that are thinner and harder.
19:49And that's what causes this pattern
19:51compression of the dermal layer. And
19:53that's how DHT plays into it. So, what
19:56about genetics, right? Where does
19:59genetics come into this story? They're
20:02certainly involved in male pattern
20:03baldness, but it's not how most people
20:05think, right? Most people think, you
20:07know, the hair follicle is genetically
20:08sensitive to DHT, but that just isn't
20:12true. It's more about a genetic
20:13predisposition. And those genetics come
20:15into the equation in terms of creating
20:17fibrosis, you know, the scalp muscles,
20:20the shape, the shape of the skull, and
20:22more.
20:25So, this is all well and good, this
20:27theory, but I'm not here to waste your
20:30time with theory, right? We're here to
20:32regrow your hair, to stop hair loss at
20:34the root cause, and to get you more hair
20:36over the rest of your lifetime, right?
20:38Sustainable hair regrowth. So, how does
20:41this theory actually play out in
20:43reality? as in what happens if you do
20:46reduce that scalp compression, if you
20:48reduce that scalp tension and if you
20:50improve the microirculation in the
20:52dermal layer. Simple question, will you
20:55regrow more hair or not? So, let's look
20:59at some examples.
21:01So, relaxing the scalp muscles. Hair
21:04loss researchers came up with an
21:06ingenious way to test this idea of scalp
21:08tension compressing the dermal layer. So
21:11what they did was inject the scalp
21:12perimeter muscles with a substance that
21:15causes flaccid paralysis. And that's
21:18just the scientific way of saying it
21:19causes the muscles to go like completely
21:22relaxed for a period of 3 to 5 months.
21:25So the kind of the nerdy way to say it
21:27is that it's a the substance is a
21:30presinaptic neurom modulator which means
21:32it stops the muscle nerve from firing.
21:34So the muscle like literally can't be
21:36used. It can't tense up at all. So they
21:39injected the substance right into the
21:41muscles that hold the scalp tight. So
21:43you can see these blue dots um is where
21:46they injected the substance
21:49and guess what happened? Those
21:51participants started regrowing hair like
21:54clockwork. So this wasn't just a one-off
21:56study, right? The same results, the same
21:59study has been repeated again and again
22:02and again now with amazing results,
22:04frankly, like incredible results.
22:07But unfortunately, the substance they
22:09use, it's incredibly expensive. It's
22:11probably costs at least $1,300 per
22:13session. So, the treatment has never
22:15been commercialized. But you can see
22:17here some of the results.
22:20They're absolutely outstanding. And this
22:21is just after one or two sessions, you
22:25know, like half an hour in the doctor's
22:26office relaxing these muscles. And you
22:29can see the crown area completely
22:31thinned in. The crown and the top area
22:34thickened up pretty significantly.
22:37crown area especially a lot of
22:39improvement and a lot of thickness
22:41improvement over here.
22:44These before and afters massive
22:46improvement in the crown. Massive
22:47improvement. So so good after just one
22:51or two sessions.
22:54So the study found basically a
22:57statistically significant increase of
22:59around 18% between baseline.
23:02So the author of the study also he had
23:06his theory about why this works so well
23:09and he says mechanistically the scalp
23:11behaves like a drum skin right with
23:13these tensioning muscles around the
23:15periphery. These muscle groups the
23:17frontalus occipitalis and per can't
23:20really say that word per aricular
23:22muscles create a tight tight scalp when
23:26chronically active. Right? So you can
23:28read this yourself you can pause the
23:29video if you want and read it. So it
23:32loosens the scalp reducing pressure on
23:34the perforating vascule thereby
23:36increasing blood flow and oxygen
23:37concentration.
23:39Blood flow may therefore be a primary
23:41determinant in follicular health. So
23:44what happens they reduce
23:46they they basically cause flaccid
23:49paralysis of these muscles. the scalp
23:51can the scalp tension gets reduced and
23:53the scalp compression also gets reduced
23:56as a result of that
23:58and it also allows more blood to flow
24:00into the top of the scalp and the result
24:02is really impressive improvement in hair
24:04growth.
24:06Okay, so keep that in mind. Let's move
24:08forward with the next section. So what
24:12about minoxidil? Monoxil was first
24:15invented as a blood pressure medication.
24:17So it widens the blood vessels allowing
24:20more blood to flow and thus reduces
24:22pressure. So when applied topically we
24:25know that minoxil increases blood flow
24:27by 200% above baseline. So that's a 3x
24:30and for about an hour after application.
24:32So in other words it dilates the blood
24:35vessels allowing more blood to fl to
24:37flow through them and the result is an
24:39increase in blood supply to the scalp
24:40and eventually hairy growth. So the
24:43problem with minoxidil is the effect is
24:45very temporary, right?
24:48And of course it's not really fixing the
24:49root cause. You're just temporarily
24:51improving the blood flow in the dermis
24:54and epidermis. Um just temporarily
24:56allowing a bit more blood to flow into
24:58the upper part of the dermal layer. But
25:00that's how and why it works. So keep
25:04that in mind. Let's move on to the next
25:06section. So PRP plateletri plasma,
25:11right? It's a procedure where the
25:13patient's blood is taken and the plasma
25:15part of the blood is enriched via
25:17centrifuge. So that enriched plasma is
25:20then injected back into the patient's
25:22scalp. So PRP is actually very very very
25:27effective.
25:28In a recent meta analysis, um it showed
25:31that an average increase of 27 new hairs
25:35per centime squared. So imagine a
25:37centime square. It's quite a small area
25:39and PRP is creating 27 new hairs. But
25:43the issue again is it's it's very
25:45expensive. Um making it a complete
25:47starter for most people. You're going to
25:49be paying like $800
25:51per session. And you need uh a few
25:53sessions in a row over about 6 months to
25:56make it really work. But again, it shows
25:59this undeniable point. More blood grows
26:03more hair. you you're literally you're
26:05literally taking your own blood and
26:07injecting it into the scalp and that's
26:09causing more hair to grow. So, I don't
26:11know in my mind like could that be any
26:13more obvious, you know, you're taking
26:15the growth factors and the sort of
26:18important part of the blood, injecting
26:20it in and boom, massive difference.
26:23Massive difference. Massive difference.
26:25So, PRP works essentially works
26:28incredibly well.
26:30So we have a big question next which is
26:35what about hair transplants right? If
26:38all this makes sense about the scalp
26:41compression scalp tension, how do hair
26:43transplants still work? Well, the hair
26:46transplant industry has made us believe
26:48that transplanted hairs are immune to
26:50DHT. But the actual truth is unless that
26:52patient takes finasteride, those
26:54transplanted hairs will start thinning
26:56after 5 years or so. And that's all
26:59whilst the hairs on the back and the
27:02sides of the head remain perfectly
27:03healthy.
27:06And we now have a study on this exact
27:08topic. Right? After 4 years only 9% of
27:12people had retained the same density and
27:14everyone else had lost density.
27:17So basically the hair transplant
27:19industry is built on this lie that
27:21transplanted follicles are immune to
27:23DHT. And I'll show you exactly why and
27:27how this lie exists uh in just a minute.
27:30So almost all hair transplant patients
27:33are required to take finasteride
27:35indefinitely. And this is to protect
27:37both the transplanted hairs and the
27:39native hairs from fibrosis. So remember
27:42that critical word that I talked about
27:43earlier.
27:45It protects finasteride protects the
27:48transplanted hairs from fibrosis. But
27:50without finasteride, those transplanted
27:52hairs, they will start thinning,
27:56you know. And these sort of posts are
27:57all over Reddit. They're all over our
27:59comments. Why have I seen multiple
28:02people say that hair transplants are not
28:03permanent?
28:05And we mentioned this point on our
28:07YouTube channel, and there are a lot of
28:08comments saying, you know, you're 100%
28:10correct. I was sold the story of no more
28:13drugs, nonsensitive DHD implanted
28:15follicles. 10 years later, it looks like
28:18So, we can go even further than
28:21this and look even deeper and say that
28:23larger graphs actually last longer than
28:25smaller ones. And that's because large
28:28graphs have more of the surrounding
28:30tissue and that surrounding tissue
28:32basically resists against the scalp
28:34compression and it resists against the
28:36fibrosis and therefore those larger
28:38graphs last longer.
28:41And another comment, I can attest to the
28:43fact that larger hair grafts last longer
28:46while smaller FUEE don't last long at
28:48all. I've had a I've had both over a
28:50period of 25 years.
28:53And if you look at a hair transplant
28:56graft, you'll see that it's it's not
28:58just this strand of hair. It's all this
29:01surrounding tissue with the hypodermis,
29:04the dermis, and the epidermis that comes
29:05with it, right? And this is the part
29:07that resists the fibrosis.
29:11It resists that scalp compression. And
29:13so it makes complete sense why the
29:15larger graphs equals more surrounding
29:18tissue equals fibrosis takes longer to
29:21set in. So if the hair transplant
29:23patient if they take finasteride then
29:26fibrosis won't take place. So these hair
29:29follicles will last but if they don't
29:32then the fibrosis will start to set in
29:34after the scalp compression and the
29:36inflammation sets in and so they'll last
29:39less long. And that's exactly what we
29:41see. You know, these smaller ones with
29:43less surrounding tissue, that will last
29:46less long before it starts to thin out
29:49than this one. And that will last less
29:51than this one. So hopefully this is all
29:54kind of making sense about how fibrosis
29:56works. And fibrosis comes from the
29:58inflammation. And the inflammation comes
30:00from the scalp compression. Okay, let's
30:02go over to this next important step.
30:06So the hair loss cascade. This is super
30:09super important. If you if you really
30:11want to understand hair loss, what's
30:12causing your hair loss at a deep level,
30:15then you need to understand the hair
30:16loss cascade. So step number one or the
30:19first thing, the root cause is the scalp
30:21tension, right? The scalp tension leads
30:24to scalp compression and some areas of
30:27the scalp are compressed more than
30:28others.
30:30This compression leads to scup
30:32inflammation and in the presence of DHT
30:35we get basically transforming growth
30:38factor beta 1 present and this
30:40transforming growth factor B1 leads to
30:43fibrosis. Fibrosis scar tissue formation
30:46in response to that chronic
30:48inflammation. Right?
30:50And because of this fibrosis this and
30:54the compression it leads to the scalp
30:56layer becoming thin hard and stiff and
30:59really importantly also it loses its
31:02layered structure. And that's exactly
31:04what we see in boarding scalps. We see
31:06that they are thinner, they're harder,
31:08they're stiffer and they've lost that
31:10layered structure and they've lost um
31:12adequate blood flow. So this leads to
31:16the reduced growth space, reduced blood
31:18flow and reduced structural integrity of
31:21the hair follicle. Then this leads to
31:24hair follicles basically they struggle
31:26to grow. Right? If you if you've got
31:27this reduced growth space um if your
31:30structural integrity um is compromised
31:33and if the blood flow is compromised
31:35then the hair follicles simply struggle
31:36to grow and they miniaturaturize with
31:38each hair cycle which is exactly what we
31:40see and that leads to ultimately male
31:43pattern baldness right hair loss that
31:45takes place in a pattern which is what
31:47we see with men. So
31:51what you can actually see is you can
31:53look at all the treatments, all hair
31:56loss treatments that we know about and
31:58you can put them on this health cascade.
32:01So let's go through them, right?
32:05So supplements, collagen, diet, stuff
32:07like that. You know, I personally I
32:10really like doing this stuff because
32:11it's going to improve your, you know,
32:13other aspects of your health anyway. You
32:15should be taking good supplements,
32:16should be taking collagen, improving
32:18your diet, right? But that basically it
32:21just gives the hair follicles that
32:22little extra boost, right? It's not
32:24really going to make a massive
32:26difference. And and that's what we see.
32:27That's what we see, right? Supplements.
32:29Yeah, it kind of helped a little bit
32:31helped, but it didn't really stop the
32:32hair loss. So, think about it on the
32:34hair loss cascade. So, moving up the
32:37hair loss cascade, you have slightly
32:38more effective treatments, right?
32:40Minoxil improves blood flow. Red light
32:42therapy LLT
32:45improves blood flow. Topical caffeine
32:46improves blood flow. PRP improves the
32:48blood flow. Right? And that's exactly
32:51what we see. These are slightly more
32:52effective than this one. Then moving up,
32:54we have micro needling. So micro
32:56needling works because it heals that
32:58scar tissue, right? It helps reduce the
33:00fibrosis and PRP
33:03uh same effect to a sim similar extent,
33:05right? It helps with fibrosis and that's
33:08why micro needling is more effective
33:11than say red light which is more
33:13effective than say a supplement. Then
33:16moving up, we have DHT blockers, right?
33:18Finasteride, dutasteride, saw palmetto.
33:21These are the most effective in terms of
33:24because they're further up the hair loss
33:26cascade, right? They stop the DHT and
33:29that stops everything going down, but
33:32these don't really grow regrow much hair
33:36and they also mess with your hormones.
33:38So, you can see this decreasing
33:40effectiveness of treatments as you go
33:42down the hair loss cascade. So hopefully
33:46that's all starting to make sense. So
33:49let's go on to the next section.
33:52How do you actually fix the root cause,
33:54right? How do you right now actually
33:56start stopping the hair loss and
33:59regrowing your hair? Well, let's go
34:01through because there's four main steps.
34:04So the first and obviously most
34:06important step is that you need to
34:08reduce this chronic tension
34:11which takes place through the muscles
34:13holding the ga. We've al already gone
34:15through that study where they reduce
34:17that muscular tension and the hair
34:18started regrowing.
34:20The muscles have become chronically
34:22tight in men with a although we don't
34:25know exactly why the muscles become
34:26chronically tight. It's obviously
34:28related to genetics and somewhat to diet
34:31and lifestyle. So your first line of
34:34defense against hair loss should be to
34:36reduce the chronic tension in the scalp
34:38so it isn't being compressed so
34:40aggressively and so that doesn't cause
34:42the whole rest of that hair loss
34:44cascade. Right? It takes time but as the
34:48tightness reduces the entire hair loss
34:50cascade improves with it. So you can see
34:52here the three main muscle groups at the
34:54front at the side and at the back. Those
34:57are the ones you want to be targeting.
35:00Okay. So moving on to step two and this
35:03is all about fibrosis, right? If you
35:05have visible hair loss from male pattern
35:07baldness, you will have fibrosis. So
35:10imagine like scar tissue like formations
35:13throughout the scalp that are the result
35:15of this chronic tension and this chronic
35:17inflammation. So you need to first stop
35:20the chronic tension and then proactively
35:22go about reversing some of that
35:24fibrosis. And this process also takes
35:27time. like you're literally remodeling
35:29the scalp tissue, right? It's similar to
35:31get getting rid of scar tissue. It's a
35:34lengthy process and you need to kind of
35:36stop what's causing it in the first
35:38place, but it's necessary if you want to
35:41make long-term sustain sustainable
35:44progress and if you want to actually
35:46reverse some of that hair loss. And this
35:48is honestly why hair loss is so
35:50difficult to reverse. is because this
35:52fibrosis has set in and it takes a long
35:55time and a lot of effort to reverse it
35:58but it is possible. Okay, so step three
36:01is block DHT. Remember that hair loss
36:03cascade we have DHT in it. We know that
36:06DHT plays a critical role in the
36:08creation of fibrosis. So since we're
36:10trying to get rid of that fibrosis,
36:12blocking the DHT will help with this
36:14because it's kind of it's kind of like a
36:16train that's moving and you kind of need
36:18to stop. you need to take away the the
36:21power behind that train and then you
36:22need to try and reverse it. Um so
36:24blocking the DHT will help you stop and
36:27reverse that fibrosis.
36:30It's not 100% essential. If you have
36:33mild hair loss, like if you're in your
36:3630s and you just have a mild receding
36:38hairline, you probably, you know, you
36:42may not need to block DHT as well if you
36:44get these first two steps right. But if
36:48you have aggressive hair loss, blocking
36:50DHT is going to be a big help. Like if
36:52you're if you are like me, if you are
36:54like me, when you started, you know,
36:56seeing the first signs of male pattern
36:58baldness at like 17 years old, then
37:02blocking DHT is going to be a massive,
37:04massive help. So our goal is to reduce
37:06the tension so that you eventually do
37:08not need to block DHT, but blocking DHT
37:10at the start will be a big help in the
37:13meantime.
37:14So step four, improve the blood flow.
37:18Right, this is the last step and this is
37:19more about reactivating the dormant hair
37:22follicles. It's about speeding up that
37:24recovery since we already fixed the real
37:27root cause. This is where those
37:29traditional options can be used based on
37:32your personal preferences, right? things
37:33like minoxidil, PRP and red light
37:36therapy, topical caffeine, they all work
37:38because they help stimulate blood flow
37:40on the surface of the scalp, but they
37:42don't fix the root cause, but they do
37:44reactivate the hair follicle growth and
37:47also more blood flow. It basically helps
37:50with point two as well, right? Reversing
37:53fibrosis
37:54um because you need that improved blood
37:57supply to help reheal these tissues.
38:03Okay, so let's go on to the next step.
38:06This is super super important and this
38:09is going to help everything else make a
38:11lot of sense. So the hair loss industry
38:15does not want this information out. Like
38:18just being bluntly honest, the hair loss
38:20industry doesn't want this information
38:21out. If you spend any time looking into
38:24the medical industry, you'll know that
38:26the real bucks, the real money is made
38:29through prescriptions and subscriptions,
38:32right? This idea I talked about at the
38:34start of the presentation, a customer
38:36for life is where the real money is
38:38made. The hair loss industry wants to be
38:41billing you every single
38:44month. To be honest, like really really
38:47try and understand this reality.
38:50The hair loss industry has zero
38:52intention to fix the root cause of your
38:54hair loss because it would simply just
38:56kill their business model's head, right?
38:59They want to build your credit card
39:01every single month up until you just
39:05finally say enough is enough.
39:07And if you want to get into the business
39:09side of it, these hair loss companies
39:11are valued on their MR, so monthly
39:14recurring revenue. Their goal, simply
39:18put, is to have millions of men plugged
39:21plugged into their monthly payment plan
39:23with no way to escape. And that's how
39:25they become worth billions of dollars,
39:28right? You know, here it is, just
39:30another hair loss brand making billions
39:32of dollars selling bog stand and minoxil
39:34and finasteride to their subscribers.
39:36You know, subscribers 2.2 million and
39:39that's why it's worth billions of
39:40dollars. this hair loss company. This is
39:43this is where they want you and this is
39:45where they wanted me and this is what
39:47this is why I was being led in the wrong
39:49direction about what actually causes
39:51hair loss because all these companies
39:53they're pushing their monthly
39:54subscriptions, their prescriptions,
39:56they're pushing on you and none of them
39:58actually fix the root cause.
40:01So ask yourself why why in the hell
40:06why in the hell would they ever threaten
40:07their cash cow by providing any kind of
40:10treatment but a monthly subscription?
40:13The absolute last thing they want for
40:14you is to fix the root cause and just
40:17pull the plug on their monthly recurring
40:19revenue model. And you know they're
40:22going to fight tooth and nail to protect
40:24their profit-making machines, right? So
40:26I hope this is making sense.
40:29So, not to get too dark, but this is the
40:34most in-depth video that I've made on
40:36this topic. And honestly, I was really
40:38hesitant and slightly worried and
40:42wondering if I should actually release
40:43it, right? I've been personally
40:46threatened in the past when I talked
40:48about this topic before because it it
40:51simply exposes an entire industry built
40:54around profit and MR.
40:57It's not built around what's actually
40:59best for the individual long term.
41:02Right? So, I've received emails like
41:03this telling me to stop talking about
41:05this topic. This is one of our videos.
41:07So, our first, you know, one of our
41:09first videos on the real cause of hellos
41:11and it got hundreds of thousands of
41:12views on YouTube. And um, you know, I
41:15was
41:17they they were trying to recommend that
41:19I not talk about this topic anymore.
41:22But the question to me is what what are
41:24they so afraid of, right?
41:28Uh it's it's super important. So
41:32look at where all the research and
41:33development dollars are going. You know,
41:35if you look at the R&D money, it's all
41:37going um towards a pay monthly product.
41:41There's literally no money to be made
41:43fixing the root cause with a you know, a
41:46simple one-off product that costs a few
41:47hundred. There's no money in that. All
41:50these all these products
41:53um all these drugs, we've talked about
41:54them on our YouTube channel. they're
41:56all, you know, pay monthly subscription
41:58products and that's because that's the
42:02way that these companies make a profit.
42:05So hopefully this is all making sense.
42:07Now, if we just go back up to the hair
42:10loss cascade, then
42:13you can basically see how all these
42:15products are down here on the hair loss
42:16cascade, right? None of them are
42:18actually fixing the root cause. So based
42:20on what we learned, there's basically
42:22one treatment that stands out above all
42:24the others. Um, you can do it at home.
42:26It costs nothing and it has zero side
42:28effects. And this is basically why the
42:31hair loss industry hates it. So, let's
42:33get into this scalp massages.
42:37Right? If you look on the hair loss
42:39cascade, the first thing is the scalp
42:41tension, which leads to the scalp
42:44compression. So, when done right,
42:47standardized scalp massages help reduce
42:49this muscular tension around the scalp
42:50perimeter. They help break down the
42:53fibrosis. step two, and they restore
42:55overall better microirculation into the
42:58scalp layers. And that was step four on
43:00on getting to the root cause. But of
43:02course, the hair loss industry doesn't
43:04want you doing them because it's
43:06completely free and it fixes the root
43:08cause.
43:09So, there have been quite a few studies
43:11on scalp massages. There haven't been
43:13the ones that we really, really want to
43:15see because, you know, keep in mind that
43:17a study is incredibly expensive. It
43:20probably costs like a million dollars or
43:22more. So why would anyone do a study and
43:27spend a mill a million bucks when they
43:29have zero financial incentive? So if
43:32you're waiting for like a really large
43:34scale perfect study on scalp massages,
43:37like honestly just forget it because no
43:39one's going to pay for that. They have
43:41literally no financial incentive.
43:43Luckily, there have been a few studies.
43:46So there was one out of 2019 where 70%
43:49of participants who tried the manual
43:51scalp massages reported positive results
43:54after about 6 months.
43:57Then there have been other scalp massage
43:59studies. Um
44:01one study showed that the hair thickness
44:03increased by about 10% after 24 weeks
44:07and that was only from 4 minutes of
44:09massage per day. So the mechanical
44:11forces of the on the scalp, they're
44:13reactivating the hair follicles through
44:15improved circulation, stimulating growth
44:17factors and breaking down that fibrosis.
44:21Right? So they used a basic massage
44:23tool. Um they only used it for 4 minutes
44:26per day and the hair thickness was shown
44:28to increase.
44:30So we know that these scalp massages do
44:32work. But there is basically one major
44:36flaw with scalp massages and that is
44:38that they take a long time. You know,
44:41you're literally aiming to remodel the
44:44scalp tissue to reduce that muscular
44:46tension on the scalp. And like as you
44:48can imagine, changing the structure of
44:50the tissue in the body. It just simply
44:52doesn't happen overnight, right? Male
44:55pattern baldness takes place over a long
44:57period of time. You can't just expect to
44:59reverse those tissue changes overnight,
45:02you know? It takes time to physically
45:04change the scalp. Just like someone
45:07starting out in the gym or at a yoga
45:09class, they can't expect to grow new
45:11muscles or become flexible overnight.
45:13You can't expect to remodel the scalp
45:14tissues and relax those scalp muscles
45:17very quickly. It does take time.
45:21But, and this is really important,
45:23there's one incredibly positive thing
45:25about this. Those changes that you make
45:28to your scalp, they tend to last. they
45:30tend to accumulate.
45:32You're physically changing the structure
45:34of the scalp and the scalp muscles. So
45:36those changes are semi semi-permanent at
45:39worst, right? You're taking your muscles
45:42out of chronic contraction. So at best,
45:45you could be making improvements to your
45:47scalp that last many years or even
45:49potentially decades. And this is exactly
45:52what one of the studies found. Simply
45:54found the longer you do it, the more
45:56effective it is.
46:00So, simply put, most people give up. And
46:04scalp massages do work, but most people
46:06simply don't do them correctly or
46:08consistently enough to start seeing a
46:10difference. And let's just be honest
46:12here, like manual scalp massages are
46:15incredibly tiring, they're boring, and
46:17they're tedious. You know, you're going
46:18like this for 10 minutes per day and
46:21considering that the study showed that
46:24we need to be doing them for about 36
46:25hours to start seeing a difference. You
46:27know, that's 10 minutes per day for 7
46:30months in a row. And this is where most
46:33people fail and then complain that
46:36massage doesn't work. They simply
46:38haven't accumulated enough hours. And
46:41when it comes to massage, it's the more
46:44the better because the changes to the
46:46muscles and the scalp tissue that
46:47accumulates over time. You know, think
46:50about the muscles holding the scalp
46:52tight. Think about that fibrosis. It
46:54takes a long time to reverse it. So, up
46:57until now, most people simply just they
46:59couldn't do it consistently enough.
47:01Right? I've personally found this. I I
47:03think I'm quite a self-disciplined
47:05person, but I couldn't do scalp massages
47:09uh manual scalp massages with my own
47:11hands. So, what is the option, right? A
47:14team of researchers and engineers from
47:16the University of Bristol and from the
47:18University of Birmingham in the UK,
47:20knowing all the things that we just
47:21talked about, all that deep research,
47:25everything about the hair loss cascade,
47:27they set about creating a new way to
47:29reverse hair loss by getting to the root
47:31cause. This is the most important point,
47:34the root cause, the scalp compression
47:35and the fibrosis. So, massages do work,
47:40but they're also just too boring, too
47:42tedious, too much, too much work. Lean
47:46basically people just to quit before
47:48they reach 36 hours. So, after 3 years
47:51of development, the team created a
47:53revolutionary new device for men with
47:55hair loss. So, this is a gamecher
47:58because it doesn't require drugs. It
48:00doesn't require a messy topical. It
48:03doesn't interfere with your hormones. It
48:04doesn't mess with your hormones. And
48:07most importantly, it's not a band-aid
48:09solution. It fixes the root cause. And
48:12it doesn't require a monthly
48:14prescription or subscription.
48:16So, the hair loss industry basically
48:19hates this device because they realize
48:21that it exposes a glaring issue with
48:25their MR business models.
48:27It's a huge issue and it's basically
48:30just they can continue to charge people
48:32monthly for the rest of their lives
48:33because their products will simply never
48:36ever fix the root cause. They're just
48:38not high enough up on the hair loss
48:39cascade. So, it's obvious to me why the
48:43hair loss industry hates this device
48:44because they make their profits by
48:46selling band-aid solutions that cannot
48:49and will not fix the root cause. And you
48:52can see some of the results that people
48:54are getting with the device. You know,
48:55crown just completely filled in.
48:58Hairline and crown filled in. Crown and
49:00top of the scalp filled in. Hairline
49:03filled in. Crown filled in. These are
49:06all results that people are getting.
49:09Massive improvement here. So, how does
49:13it work? The device works through three
49:16main mechanism. Firstly, it reduces that
49:19muscular tension around the perimeter of
49:20the scalp by gently pushing upwards and
49:23lifting upwards. It helps relax those
49:25muscles out of the chronic contraction.
49:27It's designed to fit perfectly on the
49:29right part of the scalp specifically
49:32so that this rising and falling motion
49:35focuses on the perimeter muscles.
49:38Secondly, it reverses fibrosis by
49:40pushing upwards pushes the top of the
49:43scalp together. This causes that
49:46squeezing and pinching motion that
49:48signals to the body that repair is
49:49needed to the top of the scalp. It helps
49:51break down that fibrosis and the body
49:54reheals it without without that such
49:56constrictive fibroic collagen.
49:59And then thirdly, it gently pushes more
50:02blood through the vascule of the scalp.
50:04It's just that tiny bit of extra
50:06pressure throughout the dermal layer um
50:09that helps reignite those dormant hair
50:12follicles back into growth and growing
50:15phase. And that blood blood flow
50:17improvement also helps with getting rid
50:20of fibrosis.
50:22So essentially, it's like all the
50:23benefits of a prolevel scalp massage,
50:26but without the tedious work of having
50:28to do it yourself, right? And that just
50:30simply allows you to reach this 36 hours
50:33of cumulative massage much more quickly
50:35and easily because the device was
50:38designed to target the entire scalp, the
50:41um the entire scalp all the time. And
50:43you can simply put it on, leave it to
50:46work for as long as you want and you
50:47don't have to be going like this, you
50:49know, for 10 minutes a day uh for 7
50:51months in a row.
50:54So the researchers they basically during
50:57the development phase uh they conducted
50:59a study on the device to measure blood
51:01flow in the scalp during use and the
51:04researchers measured BPU
51:07um which stands for blood fusion units
51:09in the front and top of the scalp. So
51:11what they found was the BPU start
51:13increased um after starting and
51:16continued at elevated levels for about
51:18an hour after stopping. So this
51:20basically confirmed that blood flow in
51:21the scalp is increased during use. We're
51:24currently waiting to see a large scale
51:26study that measures hair growth after
51:28using the device for 6 months. But since
51:30this kind of study costs millions of
51:32dollars to complete, that will take
51:34time. If you don't want to wait, here's
51:36what to do next. So, introducing the
51:39Grow Band is created out of three years
51:42of R&D and after 10 years of basically a
51:45relentless search into the real cause of
51:48hair loss. Importantly, it's developed
51:51with safety, convenience, and relaxation
51:53in mind.
51:55It stops hair loss at the top of the
51:57hair loss cascade, right? Super
51:59important. You need to get to the root
52:01cause if you want to win the fight
52:04against hair loss for the rest of your
52:05life. Sure, you can stop it. um in the
52:08meantime by blocking DHT with
52:10finasteride, something like that, but
52:12you're not getting to the root cause. Is
52:14that really a sustainable way to stop
52:17hair loss for the rest of your life?
52:19Right? It does away with this pay
52:21monthly business model the hair loss
52:23industry aims to extract from their
52:26customers.
52:27Importantly, it's not a quick fix. It's
52:29not a gimmick. It's simply a
52:31well-designed, effective, researchbacked
52:34tool. So tissue remodeling takes time,
52:37but those changes will last longer than
52:39all other treatments and the positive
52:41changes to your scalp accumulate over
52:43time. Super important this part.
52:46It's also the grow band is battery
52:48powered and it's programmable. So you
52:50can choose the settings you like and
52:51then you can just sit back and relax
52:55while it does all the hard work. So you
52:57can see here it pushes upwards on the
53:00sides reducing that tension through the
53:04three main muscle groups on the front,
53:06on the back, and on the sides. Then at
53:08the top of the scalp, you can see this
53:10creasing and this pinching. This is what
53:12helps reduce the fibrosis. And if you
53:15try to do this with a manual massage,
53:17you'd have to do very targeted small
53:19areas. But with this device, with the
53:22grow band, you can do basically all the
53:24top of the scalp and especially the
53:26crown. This is why people are seeing
53:28such good results on the crown area.
53:32And these are the settings that you
53:34choose. So you can inflate and deflate
53:36for as long as you want.
53:40So you can see the lifting process here.
53:42It's lifting and then it's squeezing at
53:44the top and it's also improving the
53:46blood flow into the top of the scalp.
53:50So using the grow band, it doesn't mean
53:53giving up on all other hair loss
53:54treatments, right? It's not either or,
53:56but it means fixing the root cause and
53:59it means winning the fight against hair
54:00loss in the long term. So, use basically
54:04just use the hair loss cascade chart to
54:07assess the long-term effectiveness of
54:09the different hair loss treatments that
54:10you want to use. That way, you can avoid
54:13these band-aid solutions that are just
54:15going to require a monthly payment
54:17subscription for the rest of your life.
54:19If you want to keep your hair, you're
54:20going to have to be paying for the rest
54:21of your life. There is literally no
54:22other way unless you fix through cause.
54:26So, please share this video with friends
54:29and family if they're also worried about
54:31hair loss. If you've got a friend who
54:32has hair loss or if they're worried
54:34about hair loss, share this video with
54:36them so that they can see what is
54:39actually causing the hair loss in the
54:40first place and share with them this
54:43hair loss cascade so that they know
54:45which treatments are going to be
54:46effective, where they fall on the hair
54:48loss cascade. So, thanks for watching,
54:51guys. If you do have any questions,
54:53please leave them down below. I'm going
54:55to answer every single good question.
54:57And if you've got a thought or a comment
54:59or a critique, then back up your
55:02critique with a study or some sort of
55:04fact or article or blog that we can go
55:08into and we can do an ask me anything in
55:10the next video. I'll go through all the
55:12good uh questions and we can go through
55:14that. Don't just be an NPC and comment
55:16like, oh, you know, don't just give this
55:18Helos industry NPC comment. Give an
55:21actual argument and critique so that we
55:24can go through it together and we can
55:26figure this out because this is what has
55:28worked for me and I really think it it's
55:31going to work for other people. Also,
55:33I'm going to leave this presentation, a
55:35link to this presentation down below so
55:37you can also go and check it out. Thanks
55:40guys and I'll see you in the next

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